MEDICAL FACTS: My gallbladder died-emergency removal Gallstones-My risk=11.1%=1.58x7.0%Typ. Hypertriglyceridemia [started decade ago], yet my data says I have typical odds: rs964184-CC Diagnosed as bipolar for almost two decades and meds made my high stress career liveable, but my data says I have reduced odds: rs4948418- CC
True Believers think: 1.Large segment sizes are passed intact and are proof we have detectable cousins. 2.Segments are tested and can be detected in 23andMe raw data. 3.They are doing a public service when they stalk others to proselytize and/or intimidate them with harassment. 4.I care about their opinions even when Iam mostly revolted by them. manipulative and abusive stalking of me on every post, including those where I originated the topic. 5.Consider it their duty to derail the posts and topics of with whom they disagree with personal attacks and irrelevant off topic provocations. Invisible Pink Unicorn in its Natural Habitat http://www.facebook.com/photo.php?fbid=10150538693581457 With the Hopes that our World is built on they were utterly out of touch, They denied that the Moon was Stilton; they denied she was even Dutch; They denied that Wishes were Horses; they denied that a Pig had Wings; So we worshipped the Gods of the Market Who promised these beautiful things. Rudyard Kipling-The Gods of the Copybook Headings Do not try to convince the True Believers.
Oregon, Washington, Kentucky, Arkansas, Texas, Georgia, South Carolina, North Carolina, Virginia, Scotland, Kent, England, Ireland, Germany, France, Ireland, Isle of Sky,
Father-orphaned in Denver;lived in Nebraska. Mother born Arkansas;ancestry in KY, GA, SC, NC,&Virginia.
My maternal lineage is, starting with my mother:
Noda Moore-AR Lillian Rucker-AR Susan Willie Caldoni Saffroni Little-AR Susan Childress-AR Susan Elvina Childress-GA Sarah Massey-NC Sarah Work-SC Margaret [Unknown]-VA This is a reader's digest version of my maternal lineage: http://webspace.webring.com/people/tu/um_910/MOORE.HT
Diagnosed Type II Diabetes though my data say I have average risk:
rs7903146-CT Type 2 Diabetes:My Risk=25.3%=.98x25.7% Typical risk I have T2D/Type II Diabetes and one maternal aunt who has it. It did not start until my mid-50s. I retired from 26 years of working in corrections, which kept me physically active. I have hypertriglerides and we have them under control, which may be why my mother/aunts were affected with late onset AD. Diet does not seem to work. Meds do not seem to work. I may have to exercise more.
All my med. history is documented with my health insurance company and my offspring are
few, so I am willing to participate in any research. My full genome sequence for my mtDNA is already public with the NCIB and I am an open book on my medical history.
I am diagnosed and treated for being bipolar. The True Believers who have not yet admitted and gotten treatment are a pain.
MY DNA MADE PUBLIC:
I am J1c3d yDNA and H3 mtDNA per FTDNA.These are my places for data on my DNA:
On FTDNA, my shared cM runs from 20.77cM to 66.02cM and includes 138 individuals.
23andM for the same range of 20.77cm to 66.02cM in RF, it includes 41 individuals. About Me Tiffany at 23andMe Customer Service (Tiffany B.) wrote:Dec 20, 2011 (6 hours ago) John, We have suspended your Community posting privileges effective immediately. Your recent posts are in violation of the Terms of Service agreement as well as our Community Guidelines. See https://www.23andme.com/about/tos/and https://www.23andme.com/you/community/guidelines/ Please note that you still have the ability to send and receive messages and invitations. Also, this revocation does not affect access to your data or account. Should we receive reports from other users that you are abusing the messaging system, that privilege will also be terminated immediately without notice. Let me know if you have any questions. Sincerely, Tiffany
AGT228912417rs699A or G AA
We observed significant association between tagging-SNP rs699 (M235T), located in exon 2 of the AGT gene, and AF. The AA genotype of rs699 increased the risk of AF by 70% (95% confidence interval, 1.01-2.85; P = .044) under a recessive model (AA vs AG + GG). http://www.sciencedirect.com/science/article/pii/S0022073609005378
ZFHX371586661rs7193343C or T CC
P = 1.4x10-10
A sequence variant in ZFHX3 on 16q22 associates with atrial fibrillation and ischemic stroke-A variant in the ZFHX3 gene on chromosome 16q22, rs7193343-T, associated significantly with AF (odds ratio OR = 1.21, P = 1.4 10-10).
A sequence variant in ZFHX3 on 16q22 associates with atrial fibrillation and ischemic stroke http://www.nature.com/ng/journal/v41/n8/full/ng.417.html
Atrial Natriuretic Peptide Frameshift Mutation in Familial Atrial Fibrillation http://www.ncbi.nlm.nih.gov/pmc/articles/PMC2518320/
For genotype-phenotype association analyses, we assumed an additive-allele model of inheritance and we conducted association tests using regression models with generalized estimating equations (GEE), as well as family-based association testing using FBAT. http://www.ncbi.nlm.nih.gov/pmc/articles/PMC1995607/